Brian Curl wrote:No soy. Guessing you could just get a straight shot however to taste the espresso. I am referring to espresso drinks rather than just regular brewed coffee. I've had your brewed coffee Nimbus and the roasting and deep full flavor show and is pro baseball compared to little league (mcd's brewed coffee). (never had a latte at your place)
However, when combing the steamed milk/espresso that mcd's produces compared to local shops and the gap becomes much, much less. IMO
Nimbus Couzin wrote:And of course we use ONLY organic milk at Ray's, which affects flavor slightly,
Matthew Landan
Foodie
519
Thu Mar 29, 2007 5:17 pm
331 East Market Street
Adam C
Foodie
761
Fri Jun 20, 2008 9:43 pm
Camp Taylor aka Louisville's food desert
However, the exagerated generalizations below for the most part are totally illogical and untrue.
Matthew Landan
Foodie
519
Thu Mar 29, 2007 5:17 pm
331 East Market Street
But,hey, as long as it's healthy. Right
Not sure what this has to do with your other points. There do seem to be some health benefits with coffee but that doesn't require you to go to a coffee shop. You can choose to brew your own at home.
Pages 681-697
Chuanhai Cao, John R. Cirrito, Xiaoyang Lin, Lilly Wang, Deborah K Verges, Alexander Dickson, Malgorzata Mamcarz, Chi Zhang, Takashi Mori, Gary W. Arendash, David M. Holtzman, Huntington Potter
Caffeine Suppresses Amyloid-β Levels in Plasma and Brain of Alzheimer’s Disease Transgenic Mice
Abstract: Recent epidemiologic studies suggest that caffeine may be protective against Alzheimer’s disease (AD). Supportive of this premise, our previous studies have shown that moderate caffeine administration protects/restores cognitive function and suppresses brain amyloid-β (Aβ) production in AD transgenic mice. In the present study, we report that acute caffeine administration to both young adult and aged AD transgenic mice rapidly reduces Aβ levels in both brain interstitial fluid and plasma without affecting Aβ elimination. Long-term oral caffeine treatment to aged AD mice provided not only sustained reductions in plasma Aβ, but also decreases in both soluble and deposited Aβ in hippocampus and cortex. Irrespective of caffeine treatment, plasma Aβ levels did not correlate with brain Aβ levels or with cognitive performance in individual aged AD mice. Although higher plasma caffeine levels were strongly associated with lower plasma Aβ1-40 levels in aged AD mice, plasma caffeine levels were also not linked to cognitive performance. Plasma caffeine and theophylline levels were tightly correlated, both being associated with reduced inflammatory cytokine levels in hippocampus. Our conclusion is two-fold: first, that both plasma and brain Aβ levels are reduced by acute or chronic caffeine administration in several AD transgenic lines and ages, indicating a therapeutic value of caffeine against AD; and second, that plasma Aβ levels are not an accurate index of brain Aβ levels/deposition or cognitive performance in aged AD mice.
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